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Depleting intracellular GSH with buthionine sulfoximine increases elesclomol (ES)-induced human hepatocellular carcinoma JHH7 cell death, whereas the copper chelator TTM combined with elesclomol (ES) treatment of human non-small cell lung cancer ABC1 cells does not affect their growth in the presence of elesclomol (ES), indicating that elesclomol participates in promoting a unique copper-dependent cell death, which cannot be blocked by apoptosis inhibitors (5)

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