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The resident NSCs also called type B cells are predominantly in a resting state, have a radial-glial like morphology and show formation of vascular endings with the blood vessels in the SVZ

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Collectively, these mitochondrial dysfunction-associated events contribute to ferroptosis-related nucleus pulposus cell injury and extracellular matrix degradation in IDD, thereby suggesting that targeting mitochondrial dysfunction may represent a potential therapeutic strategy for IDD

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