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Taking all this data together it seems logical or appropriate to conclude that, because IGF1-R has a different signalling pathway that can maintain lipid oxidation in the liver, FFA uptake in muscle, and activates mTORC1 could reactivate IR through tyrosine kinase activity on IRS, thus displacing serine phosphorylation, reinstituting insulin signalling

The majority of circulating UA comes from the metabolism of endogenous purine nucleotides in the liver, while approximately 20% originates from the diet 3

Rangel Moreno J, Estrada Garca I, De La Luz Garca Hernndez M, Aguilar Leon D, Marquez R, Hernndez Pando R

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PMID 1980860