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However, an exact knowledge of the SFN effect on the redox-regulation in human T-cells is crucial to estimate its clinical relevance in T-cell related diseases, since the redox balance strongly modulates T-cell functions ( Here, we show that SFN suppressed costimulation-induced activation and proliferation of primary human T-cells without having cytotoxic effects

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The medications resist being degraded by DPP-4, which is the enzyme that breaks down naturally produced GLP-1 within a few minutes

Renal cholesterol accumulation: a durable response after acute and subacute renal insults

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