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To elucidate the specific role of each putative binding site, we generated constructs with mutations in the P65 binding motif 1 (MutA), motif 2 (MutB), or both (MutAB) (Fig

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Consequently, BACH1 stabilization leads to the upregulation of key growth factors and their receptors, driving vascular endothelial cell proliferation and tube formation to promote angiogenic repair